Subscrib

Log In

TRABID inhibition activates cGAS/STING-mediated anti-tumor

TRABID inhibition activates cGAS/STING-mediated anti-tumor

Magnitude of Therapeutic STING Activation Determines CD8+ T Cell-Mediated Anti-tumor Immunity - ScienceDirect

Frontiers The interplay between autophagy and cGAS-STING signaling and its implications for cancer

ZRANB1 Is an EZH2 Deubiquitinase and a Potential Therapeutic Target in Breast Cancer. - Abstract - Europe PMC

IJMS, Free Full-Text

Delivery of STING agonists for adjuvanting subunit vaccines - ScienceDirect

ZRANB1 Is an EZH2 Deubiquitinase and a Potential Therapeutic Target in Breast Cancer. - Abstract - Europe PMC

Anti-tumor mechanism of cGAS-STING pathway.Fragments of DNA from cancer

Drug developers switch gears to inhibit STING

Ubiquitin ligase enzymes and de-ubiquitinating enzymes regulate innate immunity in the TLR, NLR, RLR, and cGAS-STING pathways

Mitotic SENP3 activation couples with cGAS signaling in tumor cells to stimulate anti-tumor immunity

TET2-mediated tumor cGAS triggers endothelial STING activation to regulate vasculature remodeling and anti-tumor immunity in liver cancer

TRABID-mediated VPS34 stabilization controls liver metabolism to

Zranb1 Mouse Gene Details, zinc finger, RAN-binding domain containing 1

Frontiers The cGAS/STING Pathway: A Novel Target for Cancer Therapy